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PIGM rabbit pAb
商品貨號(hào): PLA021123
適 應(yīng) 性: 人,小鼠,大鼠
WB IHC
¥600元
規(guī)格:
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MSDS
說明書
商品描述
  • 基因名稱: PIGM
  • 蛋白名稱: PIGM
  • Human_gene_id: 93183
  • Human_gene_link: http://www.ncbi.nlm.nih.gov/sites/entrez?db=gene&term=93183
  • Human_swiss_prot_no: Q9H3S5
  • Human_swiss_link: https://www.uniprot.org/uniprotkb/Q9H3S5/entry
  • Mouse_gene_id: 67556
  • Mouse_gene_link: https://www.uniprot.org/uniprot/67556
  • Mouse_swiss_prot_no: Q8C2R7
  • Mouse_swiss_link: https://www.uniprot.org/uniprotkb/Q8C2R7
  • Rat_gene_id: 79112
  • Rat_gene_link: https://www.uniprot.org/uniprot/79112
  • Rat_swiss_prot_no: Q9EQY6
  • Rat_swiss_link: https://www.uniprot.org/uniprotkb/Q9EQY6
  • 特異性: This antibody detects endogenous levels of PIGM at Human/Mouse/Rat
  • 組成: Liquid in PBS containing 50% glycerol, 0.5% BSA and 0.02% sodium azide.
  • 來源: Polyclonal, Rabbit,IgG
  • 稀釋: WB 1:500-2000;IHC-p 1:50-300
  • 純化工藝: The antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen.
  • 濃度: 1 mg/ml
  • 儲(chǔ)存: -15°C to -25°C/1 year(Do not lower than -25°C)
  • 說明書: 1
  • Msds: MSDS_Antibody.pdf
  • 分子量: 47kD
  • 功能: disease:Defects in PIGM are the cause of glycosylphosphatidylinositol deficiency (GPID) [MIM:610293]. GPID is an autosomal recessive trait that results in a propensity to venous thrombosis and seizures. Deficiency is due to a point mutation in the regulatory sequences of PIGM that disrupts binding of the transcription factor SP1 to its cognate promoter motif, leading to a strong reduction of expression.,function:Mannosyltransferase involved in glycosylphosphatidylinositol-anchor biosynthesis. Transfers the first alpha-1,4-mannose to GlcN-acyl-PI during GPI precursor assembly.,pathway:Glycolipid biosynthesis; glycosylphosphatidylinositol-anchor biosynthesis.,similarity:Belongs to the PIGM family.,
  • 相關(guān)產(chǎn)品: RS0001,RS0002,YM3028,YM3029
  • 細(xì)胞定位: Endoplasmic reticulum membrane ; Multi-pass membrane protein .
  • 科研貨號(hào): PLA021123
PIGM rabbit pAb
Catalog No PLA021123
Product information
  • 基因名稱: PIGM
  • 蛋白名稱: PIGM
  • Human_gene_id: 93183
  • Human_gene_link: http://www.ncbi.nlm.nih.gov/sites/entrez?db=gene&term=93183
  • Human_swiss_prot_no: Q9H3S5
  • Human_swiss_link: https://www.uniprot.org/uniprotkb/Q9H3S5/entry
  • Mouse_gene_id: 67556
  • Mouse_gene_link: https://www.uniprot.org/uniprot/67556
  • Mouse_swiss_prot_no: Q8C2R7
  • Mouse_swiss_link: https://www.uniprot.org/uniprotkb/Q8C2R7
  • Rat_gene_id: 79112
  • Rat_gene_link: https://www.uniprot.org/uniprot/79112
  • Rat_swiss_prot_no: Q9EQY6
  • Rat_swiss_link: https://www.uniprot.org/uniprotkb/Q9EQY6
  • 特異性: This antibody detects endogenous levels of PIGM at Human/Mouse/Rat
  • 組成: Liquid in PBS containing 50% glycerol, 0.5% BSA and 0.02% sodium azide.
  • 來源: Polyclonal, Rabbit,IgG
  • 稀釋: WB 1:500-2000;IHC-p 1:50-300
  • 純化工藝: The antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen.
  • 濃度: 1 mg/ml
  • 儲(chǔ)存: -15°C to -25°C/1 year(Do not lower than -25°C)
  • 說明書: 1
  • Msds: MSDS_Antibody.pdf
  • 分子量: 47kD
  • 功能: disease:Defects in PIGM are the cause of glycosylphosphatidylinositol deficiency (GPID) [MIM:610293]. GPID is an autosomal recessive trait that results in a propensity to venous thrombosis and seizures. Deficiency is due to a point mutation in the regulatory sequences of PIGM that disrupts binding of the transcription factor SP1 to its cognate promoter motif, leading to a strong reduction of expression.,function:Mannosyltransferase involved in glycosylphosphatidylinositol-anchor biosynthesis. Transfers the first alpha-1,4-mannose to GlcN-acyl-PI during GPI precursor assembly.,pathway:Glycolipid biosynthesis; glycosylphosphatidylinositol-anchor biosynthesis.,similarity:Belongs to the PIGM family.,
  • 相關(guān)產(chǎn)品: RS0001,RS0002,YM3028,YM3029
  • 細(xì)胞定位: Endoplasmic reticulum membrane ; Multi-pass membrane protein .
  • 科研貨號(hào): PLA021123
  • Hunan UPT Biotechnology Co.,Ltd
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    E-mail:service@uptbio.com
    Address:
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